Radiotherapy

Definition

 

Use of ionising radiation to damage DNA to prevent cell replication

 

Mechanism

 

Most rapidly replicating cells affected the most

 

Radiosensitive tissue

- high turnover tissue

- high blood supply

 

Give DXRT in incremental radiation

 

Produce free radical by breakup of H2O

- H2O -> H+ + OH-

- free radicals denature DNA

- damage DNA so that cell can't divide 

- cell then dies without dividing

 

Particles 

- bigger particle produces greater damage

- different particles have different RBE (Radiobiological effectiveness)

- each particle works the same way- free radical production

 

Both tumour & normal cells injured

- normal cells recover

- tumour dies

 

Need O2 for DXRT to work

- hyperbaric better

- unoperated bed better

 

Types

 

1.  Photons

 

2.  Gamma Rays 

 

Go through body / not absorbed as particles

- Cobolt 60 - Almost obsolete

- Iridium - Occasionally used in Brachytherapy

- Caesium - Used for Gynaecology tumours

- Radium - Now obsolete

 

3.  X-Rays 

- diagnostic 50-150 kv

- deep X-Ray Therapy - 300 kv

- linear accelerators - 4-24 mv

 

4. Particle beams

 

Electrons - Linear Accelerators

- particle beams via linear accelerator

- produce electron beams -> absorbed as particles

- depending on energy of beam can dial up depth of beam 

- 6MeV - 20MeV 

 

5.  Beta - Rays

- electrons given off by ionised substance

- injected locally

- Strontium, Yttrium & Samarium

 

6.  Neutron beams 

- very damaging

- experimentally are producing heavy particle beams via cyclotrons as neutron beams

 

Technique

 

Fractionation

- 1 large dose vs 60 small doses

- curative / fractionation

- palliative / minimal fractionation

 

100 Centigray = 100 Rad = 1 Gy

 

Metastasis

- 1000 Centigray in 10 doses

 

Bone Tumour

- 6000 Centigray in 30 doses

 

Curative

 

Maximum possible dose with acceptable damage to normal tissue

- 60 Gy in 30 fractions over 6/52

- equivalent to 18 Gy in one dose

- fractionation decreases late effects on normal tissue

- increased differential between tumour & tissue damage

- allows repair between treament

 

Usually given 3/52 postoperatively

- allows wound healing

- minimum delay as tumour interference activates cells in arrest phase

 

Careful planning

- multiple fields

- minimum normal tissue damage

 

Palliative

 

Short course with lower total dose

- 30 Gy in 10 fractions

- 3 CentiGy or 3 Rad or 0.03 mRad

 

Simple field set ups

- late morbidity less of an issue

- delays callus formation if pathogical fracture

- slows chondroid formation at fracture but not with osteogenesis 

- 96% local remission

 

Method of Delivery

 

External Beam

 

Brachytherapy

- old method

- place radioactive agent down tube 

- high risk to doctor giving treatment

- now use remote brachytherapy

 

Intrapoerative radiotherapy

- give dose to site at time of surgery

- give high dose with minimal local effects

 

Remote Afterloading

- pour radio-active agent down into tube

 

Timing 

 

6 Week Rule

- start radiotherapy < 6/52 after OT

 

Preoperative

- better blood supply 

- needs oxygen to effect cell kill

- shrinks tissue from neurovascular bundle

- may allow limb salvage

 

Disadvantage

- impairs healing

 

Postoperative

- usually preferred

- wait for wound to heal

- start within 6/52 otherwise repopulates with tumour cells

- also easier to identify site of tumour

 

Morbidity

 

100% side effects

 

Early 

 

Erythema / Dry desquamation / Ulceration skin

Lymphopaenia

Telangectasia

 

Myelosuppresion

 

GIT effects

 

Late

 

Skin Fibrosis

Joint Contracture

Muscle Atrophy

Lymphoedema

Hair loss

 

Chronic bone changes / fracture

 

Post Radiotherapy Changes With Stress Fracture

 

Osteoradionecrosis - eg AVN Femoral head

 

Transverse myelitis

 

Lung fibrosis

 

ST & bony hypoplasia in kids

- physeal arrest

 

Endocrine suppression

 

Infertility 

 

Skin cancers

 

Sarcomatous change

 

Specific tumours

 

Osteosarcoma

 

No indication for preoperative treatment

 

May be used for

- unresectable 

- palliation for metastasis

 

Ewing's Sarcoma

 

Very high radioresponsiveness 

- but low curability

- effective if combined with chemotherapy

- surgery & chemotherapy  have better results

 

Chondrosarcoma

 

Relatively radioresistant

 

May be used for

- recurrence

- inoperable disease

 

Myeloma

 

Effective for Plasmocytoma

Combined with chemotherapy for Multiple Myeloma

 

Soft Tissue Sarcomas

 

Indications

- doubt about surgical margins

- NV structures close to tumour

- 50 Gy DXRT  preoperatively

- 10 Gy Brachytherapy postoperatively

 

Useful to give radiotherapy preoperatively for sarcomas

- because it develops a rind around the tumour 

- makes it a lot easier to excise

- operate at about 6 weeks post radiotherapy